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Excessive release of glutamate and its binding with NMDA, -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA), or kainite (KA) receptors promotes excitotoxicity cell injury with a consequent activation of many molecular pathways, resulting in intracellular calcium (Ca 2+ ) overload 90,91 , reactive oxygen species (ROS) production, mPT, and cell swelling with brain edema 92

The separation was performed using a gradient elution with water mixed with acetonitrile

Despite being a potentially critical antioxidant, not much is known about the acute effects of CS on the GSH adaptive response and how GSH levels can fluctuate both during and between CS exposures especially in the lung ELF

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To our knowledge, this is among the first trials to comprehensively assess aescin in this context and to compare its efficacy directly with rutin while investigating adjunctive interaction with standard antithyroid medication